Practice Locations

UCHealth Diabetes and Endocrinology Clinic - Anschutz Medical Campus
1635 Aurora Ct 6th Floor, Aurora, CO 80045

720-848-2650

Neda Rasouli, MD

Endocrinology, Diabetes and Metabolism

Board Certified

Locations

Practice Locations

UCHealth Diabetes and Endocrinology Clinic - Anschutz Medical Campus
1635 Aurora Ct, 6th Floor, Aurora, CO 80045

720-848-2650

UCHealth Anschutz Outpatient Pavilion - Anschutz Medical Campus
1635 Aurora Ct Aurora, CO 80045

720-848-0000

Provider Expertise

Clinical Interest for Patients

My clinical interest include the management and treatment of patients with diabetes and metabolic disorders associated with obesity.


Care Philosophy

Every patient is a unique human being and must be provided the best individualized care.


Specialties

  • Internal Medicine
  • Endocrinology, Diabetes and Metabolism ( 2009 )

Conditions & Treatments

  • Endocrine System (Hormones)

    Adult Type 1 Diabetes, Adult Type 2 Diabetes, Lipidosis

Education & Training

Medical Schools

MD, Tehran University of Medical Siences, Iran (1995)


Residency Program

Michigan State University Program (2000)


Fellowships

University of Arkansas for Medical Sciences Program (2002)

Professional Memberships

American Diabetes Association, Member


American Federation for Medical Research, Past president


information for referring providers

Referral Contact Phone

(720) 848-2650

Referral Contact Fax

720-848-2653

Clinical Interests for Referring Providers

Diabetes and its complications, hypoglycemia, dyslipidemia, Dysglycemia

Research Interest for Referring Providers

My research interests include studying the pathophysiology of insulin resistance particularly molecular mechanisms involved in adipose tissue inflammation and lipotoxicty associated with obesity and insulin resistance. I am currently investigating the effects of different PPAR ligands on adipose tissue inflammation, particularly the number and phenotype of macrophages resident in adipose tissue. In addition, I am interested in the role of adipose tissue hypoxia in the pathogenesis of insulin resistance.